- What causes a white forelock?
- What does Piebaldism mean?
- Is white forelock dominant or recessive?
- What is Waardenburg syndrome?
- Is Poliosis dangerous?
- How do you get vitiligo?
- Are albinos bald?
- Can humans Piebald?
- Is vitiligo hereditary?
- Are Free earlobes dominant or recessive?
- Are cheek dimples dominant or recessive?
- Are chin dimples dominant or recessive?
Waardenburg syndrome: A genetic disorder that causes deafness, white forelock (a frontal white blaze of hair), a difference of color between the iris of one eye and the other (heterochromia iridis), white eye lashes, and wide-set inner corners of the eyes.
What causes a white forelock?
Piebaldism is an autosomal dominant genetic disorder with the majority of those affected having a white patch of hair near the forehead. Piebaldism may be caused by genetic mutations that affect melanin production. This leads to a lack of pigmentation in areas of the skin or hair.
What does Piebaldism mean?
Piebaldism is a rare autosomal dominant disorder of melanocyte development. Common characteristics include a congenital white forelock, scattered normal pigmented and hypopigmented macules and a triangular shaped depigmented patch on the forehead.
Is white forelock dominant or recessive?
Piebaldism is an autosomal dominant congenital leukoderma, characterized by a white forelock. Histologic studies show an absence of melanocytes in the depigmented areas of skin and normal melanocytes in the uninvolved skin (Jimbow et al, 1975).
What is Waardenburg syndrome?
Waardenburg syndrome is a group of genetic conditions that can cause hearing loss and changes in coloring (pigmentation) of the hair, skin, and eyes. Although most people with Waardenburg syndrome have normal hearing, moderate to profound hearing loss can occur in one or both ears.
Is Poliosis dangerous?
Poliosis itself is not harmful. It can, however, appear alongside other conditions that pose a health risk. For that reason, poliosis can be an early warning sign of more serious health issues. Melanoma is a skin cancer arising from the cells that provide color to the skin.
How do you get vitiligo?
Vitiligo occurs when pigment-producing cells (melanocytes) die or stop producing melanin — the pigment that gives your skin, hair and eyes color. The involved patches of skin become lighter or white.
Are albinos bald?
Albinos are rare, but common enough that almost everyone has seen one, or knows someone that has. Albinos have the characteristics of other members of their species, except that their cells are unable to produce melanin, a dark pigment that results in normal coloration in the skin, scales, eyes or hair.
Can humans Piebald?
Human piebald trait is an autosomal dominant defect in melanocyte development characterized by patches of hypopigmented skin and hair. Although the molecular basis of piebaldism has been unclear, a phenotypically similar “dominant spotting” of mice is caused by mutations in the murine c-kit protooncogene.
Is vitiligo hereditary?
Vitiligo sometimes “runs in families,” suggesting a genetic basis. However, in these families, the inheritance appears to be multifactorial, involving the interaction between multiple genes and unidentified environmental factors or triggers.
Are Free earlobes dominant or recessive?
Although some sources say that this trait is controlled by a single gene, with unattached earlobes being dominant over attached earlobes, no published studies support this view. Earlobe attachment and shape are inherited, but it is likely that many genes contribute to this trait.
Are cheek dimples dominant or recessive?
Because the inheritance pattern of cheek dimples can be unpredictable, some researchers classify them as an irregular dominant trait. This means that cheek dimples are often, but not always, inherited as a dominant trait.
Are chin dimples dominant or recessive?
A cleft chin is an inherited trait in humans, where the dominant gene causes the cleft chin, while the recessive presents without a cleft. However, it is also a classic example for variable penetrance with environmental factors or a modifier gene possibly affecting the phenotypical expression of the actual genotype.